• Alopecia
• Acniform eruptions
• Benign tumors
• Bullous diseases
• Dermoscopy
• Nevi, melanoma
• Nonmelanoma cancer
• Psoriasis and others
• Stds
• Vascular tumors
• Vulvar disease

• Herpes simplex
• Genital Herpes Simplex Introduction STD
• Genital Herpes Simplex Risk factors and Rate of Transmission
• Previous HSV type 1 infection
• Genital Herpes Simplex Primary and Recurrent Infections
• Genital Herpes Simplex Signs and Symptoms
• Genital Herpes Simplex Latency and Reactivation STD
• Genital Herpes Simplex Recurrent Infection STD
• Genital Herpes Simplex Frequency of Recurrence
• Genital Herpes Simplex Anatomic Site
• Genital Herpes Simplex Asymptomatic transmission
• Genital Herpes Simplex Prevention
• Genital Herpes Simplex Laboratory diagnosis
• Genital Herpes Simplex Serology
• Genital Herpes Simplex Testing Pregnant Women
• Genital Herpes Simplex Monogamous Couples
• Genital Herpes Simplex STD Treatment
• Genital Herpes Simplex Daily Treatment Suppressive Therapy
• Genital Herpes Simplex During Pregnancy
• Warts (HPV infection)
• Genital Warts HPV Introduction and Causes STD
• Genital Warts HPV Screening Tests
• Genital Warts HPV Interpretation of Test Results
• Genital Warts HPV The HPV DNA Test
• Genital Warts HPV Lesion and Location
• Genital Warts HPV Lesion Types STD
• Genital Warts HPV Subclinical Lesions
• Genital Warts HPV Bowenoid Papulosis STD
• Genital Warts HPV Clinical Evaluation STD
• Genital Warts HPV Differential diagnosis
• Genital Warts HPV Treatment STD
• Genital Warts HPV Treatment Approach to Therapy
• Molluscum Contagiosum
• Molluscum Contagiosum STD Introduction
• Molluscum Contagiosum Clinical manifestations
• Molluscum Contagiosum Diagnosis
• Molluscum Contagiosum Treatment
• Syphilis
• Syphilis Epidemiology STD
• Syphilis Treponema Pallidum
• Syphilis Causes STD
• Incubating Syphilis
• Secondary Syphilis STD
• Syphilis Latency
• Congenital Syphilis
• Early Congenital Syphilis
• Late Congenital Syphilis
• Syphilis Laboratory Diagnosis
• Syphilis Serologic Tests
• Syphilis Nontreponemal Reaginic Tests
• Syphilis Specific Treponemal Tests
• False-Positive Serologic Test for Syphilis
• Syphilis Treatment STD
• Chancroid
• Chancroid STD Introduction
• Chancroid Clinical presentation
• Chancroid Diagnosis
• Chancroid Treatment STD
• Chancroid Syndromic management
• Lymphogranuloma venereum
• Lymphogranuloma Venereum STD Introduction
• Lymphogranuloma Venereum Clinical features
• Lymphogranuloma Venereum Laboratory diagnosis
• Lymphogranuloma Venereum Treatment
• Donovanosis (granuloma inguinale)
• Donovanosis Granuloma Inguinale STD Introduction
• Donovanosis Granuloma Inguinale STD Clinical apperances
• Donovanosis Granuloma Inguinale STD Diagnostic tests
• Donovanosis Granuloma Inguinale STD Treatment

Lymphogranuloma Venereum Clinical features

Clinical features

The clinical course of LGV is divided into three stages. The primary stage involves the site of inoculation; the secondary stage the regional lymph nodes and sometimes the anorectum; and the tertiary or late stage affects the genitals and/or rectum.

The incubation period is 3 to 30 days. A small, painless papule appears at the site of inoculation and may ulcerate. The papule may not occur or just pass unnoticed. The lesion is seen on the prepuce or glans, and the vulva, vaginal wall or cervix. The primary lesion resolves without treatment.

The secondary stage occurs weeks later. It involves the inguinal lymph nodes, or the anus and rectum. The inguinal form is more common in men, because the lymphatic drainage of the vagina and cervix is to the retroperitoneal rather than the inguinal lymph nodes. Proctitis due to LGV is more common in women and in men who practice receptive anal intercourse, and is thought to be due to direct inoculation.

The inguinal form presents with firm, unilateral, painful inguinal and/or femoral lymphadenopathy. Adenopathy above and below the inguinal ligament forms the pathognomonic "groove sign," and is present in 10 to 20% of cases. Areas of necrosis in lymphnodes may enlarge to form abscesses, which may coalesce and break down to form discharging sinuses in about one third of patients.

In the tertiary stage chronic inflammatory lesions may lead to scarring in both the eye and genital tract. Untreated LGV can lead to fibrosis and cause lymphatic obstruction, causing elephantiasis of the genitalia. Rectal involvement can lead to the formation of strictures and fistulae. These conditions are more common in women, and can give rise to the syndrome of esthiomene a Greek word meaning "eating away." There is widespread destruction of the external genitalia.