Dermnet Videos
Alopecia Videos
- Alopecia areata – Causes and Associated Conditions
- Alopecia Areata Information
- Alopecia Areata Treatment
- Course of Sudden Hair Loss Telogen Effluvium
- Evaluation and Treatment of Sudden Hairloss Telogen Effluvium
- Female Pattern Baldness and Hair Loss Causes
- Female Pattern Baldness and Hair Loss in Women
- Female Pattern Hair Loss Evaluation and Testing
- Female Pattern Hair Loss Treatment
- Hair loss and Alopecia Introduction
- Hair Loss Due To Hair Pulling – Trichotillomania
- Hair Loss Treatment and Male Pattern Baldness Medicine
- Loose Anagen Hair Syndrome
- Male Patern Baldness Causes and Hair Loss
- Male Pattern Baldness and Hair Loss Information
- Sudden Hair Loss Telogen Effluvium
- Traction Alopecia Hair Loss
- Traction Alopecia Hair Loss Treatment
- Central Centrifugal Cicatricial Alopecia
- Discoid Lupus Erythematosus – Clinical
- Discoid Lupus Erythematosus – Histology
- Discoid Lupus Erythematosus – Treatment
- Follicular Degeneration Syndrome
- Folliculitis Decalvans – Clinical
- Folliculitis Decalvans Treatment
- Hair Loss Alopecia With Scarring Information
- Lichen Planopilaris
- Lichen Planopilaris – Clinical Features
- Lichen Planopilaris Treatment
Video Topics
Discoid Lupus Erythematosus – Clinical
Discoid lupus erythematosus (DLE) is a form of chronic cutaneous lupus erythematosus characterized by hyperkeratotic inflammatory plaques which eventuate into areas of scarring. Only 5% of DLE patients go on to develop systemic lupus erythematosus (SLE). However, 25% of SLE patients have discoid lesions. DLE lesions occur most commonly on the head and neck. Patients with generalized DLE lesions involving the skin above and below the neck are more likely to develop SLE. Scalp lesions occur in 60% of DLE patients and usually result in scarring alopecia. DLE lesions are red to purple plaques with hyperkeratotic scale.
There are follicular hyperkeratotic plugs causing a carpet tack appearance as they project from the undersurface of the scale when it is removed from advanced lesions. The dyspigmentation of older lesions often presents as central hypopigmentation and peripheral hyperpigmentation. Patients may have scalp lesions only or may have concomitant lesions on the face, neck, and especially the external ears.